Max in WT synaptoneurosomes, suggesting that Src signaling may be downregulated in KI synapses. On the flip side, our power to rescue SERT functionality in KI midbrain synaptoneurosomes because of the inhibition of FAK implies elevated FAK signaling downstream of the Pro32Pro33 mutant, as verified by enhanced pFAK localization in https://courtneyx086bny7.blogaritma.com/profile